Traduction de «acute effect » (Anglais → Français) :

acute effect [ acute toxic effect ]

Definition: This block contains a wide variety of disorders that differ in severity and clinical form but that are all attributable to the use of one or more psychoactive substances, which may or may not have been medically prescribed. The third character of the code identifies the substance involved, and the fourth character specifies the clinical state. The codes should be used, as required, for each substance specified, but it should be noted that not all fourth character codes are applicable to all substances. Identification of the psychoactive substance should be based on as many sources of information as possible. These include self-report data, analysis of blood and other body fluids, characteristic physical and psychological symptom ...[+++]s, clinical signs and behaviour, and other evidence such as a drug being in the patient's possession or reports from informed third parties. Many drug users take more than one type of psychoactive substance. The main diagnosis should be classified, whenever possible, according to the substance or class of substances that has caused or contributed most to the presenting clinical syndrome. Other diagnoses should be coded when other psychoactive substances have been taken in intoxicating amounts (common fourth character .0) or to the extent of causing harm (common fourth character .1), dependence (common fourth character .2) or other disorders (common fourth character .3-.9). Only in cases in which patterns of psychoactive substance-taking are chaotic and indiscriminate, or in which the contributions of different psychoactive substances are inextricably mixed, should the diagnosis of disorders resulting from multiple drug use (F19.-) be used. | Modifiers The following fourth-character subdivisions are for use with categories F10-F19: Code Title .0 Acute intoxication A condition that follows the administration of a psychoactive substance resulting in disturbances in level of consciousness, cognition, perception, affect or behaviour, o ...

acute respiratory effect

acute lymphoblastic leukemia | ALL | acute lymphoblastic leukaemia | acute lymphatic leukemia | acute lymphatic leukaemia | acute lymphocytic leukemia | acute lymphocytic leukaemia | acute lymphoid leukemia | acute lymphoid leukaemia | acute lymphogenous leukemia

acute autoimmune neuropathy | acute idiopathic polyneuritis | acute idiopathic polyradiculoneuritis | acute inflammatory demyelinating polyneuropathy | acute inflammatory demyelinating polyradiculoneuropathy | acute inflammatory polyneuropathy | acute inflammatory polyradiculoneuropathy | Guillain-Barré syndrome | Guillain-Barré-Strohl syndrome | infectious polyneuritis | Landry-Guillain-Barré Syndrome | Landry's paralysis | AIDP [Abbr.] | GBS [Abbr.] | LGBS [Abbr.]

acute lymphoblastic leukemia [ ALL | acute lymphatic leukemia | acute lymphocytic leukemia | acute lymphoid leukemia | acute lymphogenous leukemia ]

acute myeloblastic leukaemia | acute myelogenous leukaemia | acute myeloid leukaemia | acute nonlymphocytic leukaemia | AML [Abbr.] | ANLL [Abbr.]

Blast effects Exposure to ionizing radiation from nuclear weapon Fireball effects Heat Other direct and secondary effects of nuclear weapons

Definition: A heterogeneous group of disorders characterized by the acute onset of psychotic symptoms such as delusions, hallucinations, and perceptual disturbances, and by the severe disruption of ordinary behaviour. Acute onset is defined as a crescendo development of a clearly abnormal clinical picture in about two weeks or less. For these disorders there is no evidence of organic causation. Perplexity and puzzlement are often present but disorientation for time, place and person is not persistent or severe enough to justify a diagnosis of organically caused delirium (F05.-). Complete recovery usually occurs within a few months, often ...[+++] within a few weeks or even days. If the disorder persists, a change in classification will be necessary. The disorder may or may not be associated with acute stress, defined as usually stressful events preceding the onset by one to two weeks.

Potential exposure of bystanders and residents to pesticides via the air might constitute an exposure route, which needs further attention by research and possibly also regulatory measures. Risks to human health and the environment consist of acute and/or chronic adverse effects on humans and on non-target species. Acute effects are mainly due to the high toxicity of certain PPPs. Chronic effects which might also affect the fitness of exposed populations include those due to bioaccumulation and persistence of substances, irreversible effects such as carcinogenicity, mutagenicity and genotoxicity, or adverse effects on the immune or endoc ...[+++]rine systems of mammals, fishes or birds. It has to be noted that so far no active substance, which is classified in category I for any of these effects, has been included in Annex I of Directive 91/414/EEC, certainly not if such effects are to be expected at environmental concentrations and exposure conditions.

This is a very valuable concept for us, because by using that comparative approach to link a new food entering the marketplace to a food with which we have a long history of familiarity, what we achieve is a mechanism by which we can then in comparison establish the safety of the food not in terms of absolute terms, because there is no such thing as zero risk or absolute safety, but instead it allows us to benefit from the history of safe use and therefore gives us a mechanism by which we can predict not only potential acute effects of a particular food but also the long-term effects of that food, based on our knowledge of the long-term ...[+++]effects of its traditional counterpart.

Similarly, a comprehensive meta-analysis—that's an analysis of several studies wrapped into one—on the non-acute effects of cannabis found a small but discernible residual effect of cannabis use in only two of eight cognitive domains and concluded that, based on the extant data, they “failed to reveal a substantial, systematic effect of long-term, regular cannabis consumption on the neurocognitive functioning of users”.

acute effects such as acute toxicity, irritation and corrosivity.